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  2. Make These Vegan Tempeh Spring Rolls With Peanut Sauce Ingredients Tempeh: 1/2 block tempeh (113g) thinly sliced 1/4 cup coconut aminos (60ml) 2 tsp chili garlic sauce (5ml) 2 tsp minced ginger (5g) 1 tsp minced garlic (3g) Peanut Butter Sauce: 1/4 cup peanut butter 3 tbsp coconut aminos 45ml 2-3 tbsp water 1 tbsp sriracha 15ml 1 tsp seasoned rice vinegar 5ml 1 tsp agave nectar 1/4 tsp garlic powder 1/8 tsp ginger powder Summer Rolls: Vietnamese rice paper (six) lettuce 36g Shredded Red cabbage 125g 2 sliced Green onions sliced Yellow bell pepper 120g grated carrots 120g cucumber batonnets (sliced lengthwise) 180g Instructions Tempeh: Slice the half tempeh block in thin slices lengthwise. In a small bowl, combine the tempeh and the marinade ingredients. Mix to coat the tempeh well. Heat a pan on medium-low heat, add the tempeh and the rest of the marinade to the pan. Cook until the tempeh is browned on both sides. About 10-15 mins. Peanut Butter sauce: Combine ingredients and whisk until smooth. Set aside. Summer Rolls: Fill a large bowl with water. Immerse one wrapper in the water to soften. Pull it out, being careful so it doesn't collapse on itself, and lay it on a cutting board. Fill the wrapper with the veggies, a few pieces each, add three slices of tempeh. Fold the top and bottom of the wrapper over the filling. Fold the left&right sides, and roll tightly. Repeat with remaining rice papers. Makes six rolls. Slice in half to serve, if desired. https://www.livekindly.co/vegan-food/ kalip
  3. Drug Combos May Be Advance Against Heart Failure Many patients with heart failure might live years longer if they were on a combination of newer medications, a study suggests. Researchers estimate that if certain heart failure patients were prescribed a four-pill regimen -- including three recently proven therapies -- they could live up to six years longer, compared with the regimen patients commonly use. The findings, published online recently in the medical journal The Lancet, are projections, not guarantees. But they do make a strong case for the medication cocktail, said lead author Dr. Muthiah Vaduganathan, a cardiologist at Brigham and Women's Hospital in Boston. "Combined use of these four pills would be anticipated to keep patients out of the hospital and alive for more years," he said. Heart failure is a chronic condition in which the heart muscle cannot pump blood efficiently enough to meet the body's demands -- causing symptoms such as fatigue, breathlessness and leg swelling. The new study focused on patients who had heart failure with reduced ejection fraction, which refers to how much blood the heart pushes out with each contraction. Reduced ejection fraction affects about half of heart failure patients. For a long time, the standard medication regimen for those patients was a beta blocker plus an ACE inhibitor or an angiotensin II receptor blocker (ARB). All three drugs lower blood pressure and ease the heart's workload through different mechanisms. But more recently, clinical trials have found that three other drug types can help patients live longer, beyond standard drugs alone. One is a medication called Entresto, which combines the ARB valsartan with another drug, sacubitril. Another is a diabetes drug called dapagliflozin (Farxiga), which was recently shown to benefit heart failure patients with or without diabetes. The third drug class is actually an old one: mineralocorticoid receptor antagonists, which include spironolactone and eplerenone. They help control blood pressure by blocking a hormone called aldosterone. Despite the positive findings, though, many doctors still do not prescribe those drugs for heart failure, Vaduganathan said. And there is little evidence on what would happen if patients used all three types of medication. "The optimal combination really hasn't been studied," Vaduganathan said. So his team tried to estimate the benefits of taking the newer therapies, together, over many years. To do that, they combed shorter-term data from three large clinical trials that tested the drugs against standard treatment. Then they made some projections: What would happen if patients started on a beta blocker plus the three drug classes, instead of an ACE inhibitor or ARB -- and stuck with the regimen for the long haul? For a 55-year-old, the researchers estimate, that could translate into an extra six years of life, and eight years free of hospitalisation for heart failure. For an 80-year-old, the benefits would be smaller but still important -- an extra year of life, on average, and nearly three extra years out of the hospital. In an "ideal world," all eligible patients would be on the medications, said Dr. Mary Norine Walsh, past president of the American College of Cardiology. "But here's the rub," she added. "Two of these drugs [Entresto and Farxiga] are not available as generics." So, many patients may be unable to afford the pricey medications, even with insurance. Farxiga costs roughly $500 for 30 pills, while Entresto costs about $600 for 60 pills, according to an online search. Beyond cost, not all patients should be on the drugs. Walsh, who was not involved in the study, stressed that the findings apply only to patients with reduced ejection fraction -- which leaves out half of people with heart failure. And any one patient, she said, might have other health issues that are contraindicated for a given drug. "There may be reasons not to take a medication," Walsh said. "But that shouldn't stop patients from asking their doctor questions." Specifically, she said, patients who are still on an ACE inhibitor or ARB can ask their doctor why they have not been switched to Entresto -- which guidelines say is the preferred choice. The diabetes drug Farxiga was only just approved for treating heart failure with reduced ejection fraction, Vaduganathan noted. So, it's not yet included in treatment guidelines. The study received no funding, but the researchers report connections to various drug companies that make heart failure medications. More information The American Heart Association has more on heart failure treatment. Healthday. kalip
  4. Another COVID-19 vaccine candidate has achieved good early results, a research team in China reported Friday. The experimental vaccine produced important signs of immune response in a small group of adults, a sign that it could potentially protect people against COVID-19, researchers reported May 22 online in The Lancet medical journal. "These results represent an important milestone," researcher Wei Chen, part of a team with the Beijing Institute of Biotechnology, said in a journal news release. "However, these results should be interpreted cautiously," Chen continued. "The challenges in the development of a COVD-19 vaccine are unprecedented, and the ability to trigger these immune responses does not necessarily indicate that the vaccine will protect humans from COVID-19." This particular vaccine candidate used a weakened common cold virus called an adenovirus to safely deliver COVID-19 coronavirus genetic material into human cells. This causes the body to produce the "spike" protein that the coronavirus uses to invade human cells. In turn, the immune system responds to the presence of the protein by creating antibodies that potentially could fight off future coronavirus infections, the researchers said. Within a month, most of the 108 healthy adult participants in the phase 1 trial had a fourfold increase in binding antibodies. These antibodies can bind to the coronavirus, but don't necessarily attack it. Half of the participants who received low or moderate doses and three-quarters of those who received a high dose also developed neutralising antibodies, which can protect the body against the coronavirus, researchers reported. "Not only did the virus induce antibodies but also T-cell immunity, which will also be important for confirming protection," said Dr. Amesh Adalja, a senior scholar with the Johns Hopkins Centre for Health Security, who called the trial results "very welcome." However, Adalja said the vaccine's use of a common cold virus to deliver its genetic payload could hamper its effectiveness. "Adenoviruses are very common and there are many people with pre-existing immunity," Adalja said. "In a prior trial using this type of platform for HIV vaccination, there was a paradoxical increase in risk of infection in those who are vaccinated who had pre-existing adenovirus 5 immunity. Whether this phenomenon is present with this vaccine will be important to investigate in detail in phase 2 and phase 3 trials." There was some evidence of this in the clinical trial, with people who had existing cold virus immunity showing a reduced immune response to the vaccine, the Chinese researchers noted. A full-fledged phase 2 trial has been initiated in Wuhan to see if the results can be replicated in 500 healthy adults, which would include 250 volunteers given a moderate dose, and another 250 given either a low dose or a placebo. The U.S. Department of Health and Human Services has entered a full-court press to develop potential COVID-19 vaccine candidates. U.S. federal officials said Thursday the agency would provide up to $1.2 billion to the drug company AstraZeneca to develop a potential coronavirus vaccine from a lab at the University of Oxford in England. The fourth, and largest, vaccine research agreement funds a clinical trial of the potential vaccine in the United States this summer with about 30,000 volunteers, the New York Times reported. The goal? To make at least 300 million doses that could be available as early as October, the HHS said in a statement. However, many experts have said that the earliest an effective, mass-produced vaccine would be available won't be until sometime next year, and billions of doses would be needed worldwide. The United States has already agreed to provide up to $483 million to the biotech company Moderna and $500 million to Johnson & Johnson for their vaccine efforts. It is also providing $30 million to a virus vaccine effort led by the French company Sanofi, the Times reported. More information The U.S. Centres for Disease Control and Prevention has more on COVID-19. https://consumer.healthday.com/ kalip
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  6. SPICY TOFU AND MEATY MUSHROOM VEGAN KOREAN BBQ STIR-FRY Ingredients 2 white potatoes peeled and cubed 1 cup of soy sauce 1/4th white onion diced 1/2 cup freshly chopped cilantro 1 cup of snow peas 3 cups pre-cooked brown rice ½ red pepper diced ½ green pepper diced ½ poblano pepper diced 2 cups broken up broccoli florets 1 carrot diced ½ cup white button mushrooms 1 pack of firm tofu drained and cubed 2 tablespoons of coconut oil ½ cup of slivered almonds Korean BBQ Sauce Ingredients: 1 cup soy sauce 1/2 cup brown sugar 1 tablespoon of rice vinegar 1 clove of garlic 2 table spoons of toasted sesame oil 1 tablespoon black pepper 1 teaspoon tapioca flour 1 teaspoon ground ginger Instructions Combine ingredients to make Korean bbq sauce into blender and blend until smooth. Put chopped potatoes and tofu in a large mixing bowl. Add 1 cup of Korean bbq sauce and ¼ cup chopped cilantro. Massage mixture into potatoes and tofu vigorously for 5 minutes. Let stand for another 10 minutes. Sauté potato and tofu mixture for 10 minutes until browned on each side. Add in the rest of your pre-cut veggies. Then sauté with coconut oil, 1 cup of soy sauce and the remainder of your Korean BBQ mixture until veggies are tender yet firm. Spoon 1 cup of stir fry on you plate and top with a tablespoon of silvered almonds. Nutrition Facts Spicy Tofu and Meaty Mushroom Vegan Korean BBQ Stir-Fry Serves: 8 Amount Per Serving Calories 162 % Daily Value* Total Fat 9.5 grams 14.6% Saturated Fat 0 Trans Fat Cholesterol 0 Sodium 329 milligrams 13.7% Total Carbohydrate 11 grams 3.7% Dietary Fiber 0 Sugars 3.5 grams Protein 12 grams Vitamin A Vitamin C Calcium Iron * Percent Daily Values are based on a 2,000 calorie diet. Your daily values may be higher or lower depending on your calorie needs. https://www.livekindly.co/vegan-food/vegan-recipes/ kalip
  7. As some countries ease up, others are reimposing lockdowns amid a resurgence of coronavirus infections As many parts of the world, including the United States, explore ways to ease restrictions aimed at containing the spread of the coronavirus, countries that had already opened up are closing down again after renewed spikes in infections. Such a resurgence of cases had been widely predicted by experts, but these increasing numbers come as a sobering reminder of the challenges ahead as countries chafing under the social and economic burdens of keeping their citizens indoors weigh the pros and cons of allowing people to move around again. Lebanon on Tuesday became the latest country to reimpose restrictions after experiencing a surge of infections, almost exactly two weeks after it appeared to have contained the spread of the virus and began easing up. Authorities ordered a four-day, near-complete lockdown to allow officials time to assess the rise in numbers. The re-emergence of coronavirus cases in many parts of Asia is also prompting a return to closures in places that had claimed success in battling the disease or appeared to have eradicated it altogether, including South Korea, regarded as one of the continent’s top success stories. South Korea last week rescinded a go-ahead for bars and clubs to reopen after a spike in cases, hours after officials announced the lifting of previous social distancing restrictions and the start of a “new everyday life with the coronavirus.” South Korean President Moon Jae-in warned his country Sunday to “brace for the pandemic’s second wave,” calling the battle against covid-19 a “prolonged” fight. In the Chinese city of Wuhan, where the pandemic first emerged, authorities on Tuesday ordered the testing of all 11 million inhabitants after a cluster of six new infections emerged, five weeks after the city had apparently rid itself of the disease. Germany, which is widely regarded as the model in Europe of a balanced coronavirus response, is warning that some areas may have to reinstate restrictions after localised outbreaks caused a rise in cases. “We always have to be aware that we are still at the beginning of the pandemic,” German Chancellor Angela Merkel cautioned last week as a cluster of new cases in a meatpacking plant raised fears of an intensified outbreak. “And there’s still a long way in dealing with this virus in front of us.” Some countries are going ahead with plans to lift restrictions despite evidence that cases are on the rise and the disease is far from being contained. India and Russia eased their restrictions Tuesday even as the number of infections in both countries continued to soar. Iran, the epicentre of the disease in the Middle East, with more than 110,000 reported cases, has ordered a county in the southwestern province of Khuzestan to reimpose a lockdown after cases spiked there. But the government is still planning to proceed with the reopening of schools later this week, despite a marked jump in new infections since restrictions were eased in late April. The new spikes underscore the question of when — or whether — it will ever be fully safe for coronavirus-stricken countries to lift their lockdowns. The latest cluster in Wuhan demonstrates how hard it will be to measure whether any location is truly free of coronavirus. The new cases there suggest the virus can flare up in patients up to 50 days after they have apparently recovered, said Wu Zunyou, chief epidemiologist at the Chinese Centre for Disease Control and Prevention, in an interview with state broadcaster CCTV. “The course of disease could last 30 to 50 days for some patients,” Wu said. “The virus could take longer to manifest itself in patients with weak immunity, who are also prone to ‘ons’ and ‘offs’ of symptoms.” He sought to reassure citizens that the new cases did not represent a new wave of the pandemic. “There will not be a new minor peak,” Wu said. “We have had the epidemic under control after more than three months of efforts and accumulated considerable experience in both diagnosis and [epidemic] notification. Therefore, we will not allow scattered cases to develop into massive outbreaks.” Firass Abiad, who oversees coronavirus efforts at the Rafik Hariri University Hospital in Beirut, Lebanon’s main government hospital, said a country’s success will depend less on how it curtails the spread of disease during a lockdown than on how the country manages the inevitable resurgence after lockdowns end. “A lockdown is a means and not an end,” he said. “It’s a means either to allow you to regain control or put measures in place to control coronavirus when it comes back. When we eased the lockdown, we knew there would be an increase in the number of cases.” Lebanese government officials said the lockdown was being reimposed for four days starting at midnight Wednesday to allow the authorities time to conduct contact tracing and isolation for several new clusters that have broken out in different parts of the country. They represent 104 cases in the past four days, a big jump in a small country after infections had stabilised at the rate of one or two new cases a day. Most are linked to some of the thousands of Lebanese who have been repatriated in recent weeks from around the world, said Souha Kanj, who heads the infectious diseases department at the American University of Beirut. All the returnees are tested for the coronavirus and required to quarantine for two weeks even if they are negative. But some have not been adhering to the requirement, she said, citing the case of a man who returned from Nigeria and then held a party for his relatives, infecting at least 10 of them. One was a member of the security forces, who has in turn infected a dozen or so of his colleagues. There may also be cases of renewed local transmission, perhaps stemming from people who were asymptomatic but have been silently spreading the disease, Abiad said. Lebanese released from nearly six weeks of lockdown last month have surged onto the streets, openly defying some of the continued restrictions and social distancing rules that remain in force. Similar scenes have occurred in other countries that are ending closures. Crowds of Parisians gathered on the banks of the Seine to toast their release Monday from six weeks of one of the world’s toughest confinements, prompting police on Tuesday to ban the consumption of alcohol in the vicinity of the river. “In any country where people don’t adhere to social distancing, this is going to happen. You are going to have a surge in cases for sure,” Kanj said. Correction: A line in the story about the increase of cases in France was removed because it referred instead to fatalities. https://www.washingtonpost.com/ kalip
  8. coronavirus attacks the entire body — damaging the brain, kidneys and more While the coronavirus is known to cause deadly respiratory problems, research is showing the multitude of ways the disease can ravage the entire body. Through a growing number of studies, reports and doctors’ experiences, the deadly virus has been linked to issues in everything from the brain to the toes. Here is a breakdown of the ways COVID-19 can affect different parts of the body: Eyes The coronavirus has been observed to turn eyes red, causing pinkeye, or conjunctivitis, in some patients. Physicians have suggested that the condition develops in the severely ill, and one study of 38 hospitalised patients in Hubei, China, found that a third had pinkeye. Brain Neurological symptoms such as dizziness, headaches, impaired consciousness and skeletal-muscle injuries have been documented among cases. Chinese doctors in April published a study on nervous system function in the Journal of the American Medical Association that found that patients can also experience more serious issues — including seizures and stroke, which occurs when a blood clot reaches the brain, according to the Wall Street Journal. Heart Doctors have seen alarming cases of myocarditis — an inflammation of the heart muscle — in addition to irregular heart rhythms that can lead to cardiac arrest in coronavirus patients, according to the Washington Post. “They seem to be doing really well as far as respiratory status goes, and then suddenly they develop a cardiac issue that seems out of proportion to their respiratory issues,” Mitchell Elkind, a Columbia University neurologist and president-elect of the American Heart Association, told the outlet. Sensory system The loss of the ability to taste and smell has emerged as a peculiar symptom strongly associated with the virus. The condition, which is known as anosmia, was not initially recognized as a symptom of the virus, but data from a symptom-tracking app in one study found that 60 percent of people who tested positive reported losing their senses of smell and taste, according to researchers from King’s College London. About a quarter experienced the strange symptom before developing other conditions, suggesting it may be an early sign of the virus. Blood The coronavirus also causes blood thickening and clots in the veins, according to doctors. The clots can break loose and travel to the lungs and brain, potentially causing a deadly condition known as pulmonary embolism. It’s still not known why the virus causes the blood clots to form, or why the body is unable to break them up. Gastrointestinal system Digestive issues such as diarrhoea, vomiting and abdominal pain have been among the chief complaints of many patients. Nearly half of the virus patients admitted to the hospital in the central Chinese province of Hubei, where the outbreak occurred, reported experiencing digestive problems, according to a study published in the American Journal of Gastroenterology. Kidneys Kidney damage has been reported among hospitalised patients, with an alarming percentage who require dialysis. Nearly half of hospitalised patients have blood or protein in the urine, suggesting there is early damage to the kidneys, one nephrologist told the Washington Post. And early data showed that between 14 and 30 percent of intensive care patients in New York and Wuhan, China, lost kidney function and required dialysis or continuous renal replacement therapy, according to the report. Feet Foot sores nicknamed “COVID toes” have been documented as an odd symptom linked to the virus. Physicians have observed the purple foot sores, which are similar to those of chickenpox or measles, among mostly young coronavirus patients in Italy, France and Spain. Immune system Physicians have found in some cases that a patient’s immune system goes into overdrive to fight the infection. The response, which is known as “cytokine release syndrome,” can cause damage that results in severe inflammation and organ failure, NPR reported. Lungs COVID-19 is well known to cause coughing, trouble breathing and, in some cases, severe pneumonia. The infection can work its way into the lungs, filling tiny air sacs with cells and fluid that prevent the flow of oxygen, the Guardian reported. When the air sacs become inflamed, pneumonia can develop in the lungs, which then struggle to get enough oxygen to the bloodstream — reducing the body’s ability to take in oxygen and get rid of carbon dioxide, according to the outlet. Ultimately, this can lead to death in severe cases, the outlet said. https://nypost.com/coronavirus/ kalip
  9. FDA Approves Drug for Heart Failure The FDA has approved a new use for the drug dapagliflozin (Farxiga) -- to reduce the risk of a hospital stay or death in people who have a type of heart failure. People with heart failure are more likely to get complications and early death, even when they take other drugs for heart disease such as ACE inhibitors, beta-blockers, and angiotensin receptor blockers. Farxiga's new approval is to treat heart failure with reduced ejection fraction, which means the left ventricle, or lower chamber, of the heart is too weak to pump blood out to the body very well. “Heart failure is a serious health condition that contributes to one in eight deaths in the U.S. and impacts nearly 6.5 million Americans,” Norman Stockbridge, MD, PhD, director of the Division of Cardiology and Nephrology in the FDA’s Centre for Drug Evaluation and Research, says in a news release. The approval is good news for people with heart failure, says Wilson Tang, MD, a heart failure specialist and director of heart failure research at Cleveland Clinic's Heart & Vascular Institute. "This is an area where we have had few significant landmark studies and approvals over the past decade." Farxiga belongs to a class of diabetes drugs called sodium-glucose cotransporter-2 (SGLT2) inhibitors, which also includes canagliflozin (Invokana), empagliflozin (Jardiance), and ertugliflozin (Steglatro). All four drugs are already FDA-approved to treat type 2 diabetes. They lower blood sugar by stopping the kidneys from absorbing extra sugar in the urine back into the blood. In October 2019, the FDA also approved Farxiga to reduce the risk of a hospital stay for heart failure in people with type 2 diabetes plus heart disease, or risks for heart disease. Discovery of a Heart Failure Benefit Researchers discovered the benefits of SGLT2 inhibitors for treating heart failure somewhat by accident, while studying the effects of these drugs in people with type 2 diabetes. In four large studies, people with type 2 diabetes who took an SGLT2 inhibitor were 25% to 35% less likely to be hospitalized for heart failure -- whether or not they had heart failure at the start of the study. Heart failure is an early and the most common heart-related complication in people with diabetes. The new approval is based in part on a study called DAPA-HF, which was the first to look at the effects of an SGLT2 inhibitor specifically on heart failure. This study randomly assigned more than 4,700 people who had heart failure to take Farxiga or an inactive pill (placebo) along with their regular heart failure drugs. After just over 18 months, when Farxiga was added to other heart failure medications, there was an 18% decrease in the risk of death from heart disease and a 17% lower risk of death from all causes. The drug also led to a 26% combined lower risk for a hospital stay due to heart failure, worsening heart failure, or heart-related death. The drug slowed heart failure from getting worse in people both with and without diabetes. How Does It Treat Heart Failure? "That's a great question. No one's 100 percent sure. There are lots of different theories," says Deepak Bhatt, MD, executive director of interventional cardiovascular programs at the Brigham and Women's Hospital Heart & Vascular Centre. (Bhatt has received funding from AstraZeneca, Farxiga's maker, as well as from Sanofi-Aventis, for his research on SGLT2 inhibitors.) "The simplest theory is that it's just a really good diuretic -- a medicine that removes fluid from the body," he says. "People with heart failure are often on a diuretic, also called a water pill, to get extra fluid out of their body." As the drug removes sugar from the urine, it also flushes out salt and fluid. It's likely that other mechanisms are also involved. The drug might increase ketone bodies -- chemicals our bodies make when they break down fats to use for energy. Ketones might improve the heart's energy usage. Or Farxiga and other drugs like it might protect heart muscle cells from more damage. Bhatt says it will take some time and further research to figure out exactly how Farxiga works to treat heart failure. "I think it could take years to sort it all out." Side Effects Genital yeast infections, dehydration, and urinary tract infections (UTIs) are the most common side effects, which happen as Farxiga puts extra sugar into the urine. "When urine has more glucose, there is a higher likelihood for patients to get a UTI," says Tang. Your doctor can check you for these infections if you take Farxiga, and treat you with an antifungal or antibiotic drug if you need it. You may not be able to take Farxiga if you have severe kidney disease or you're on dialysis, because it could affect how your kidneys work. Your doctor might do tests to check how well your kidneys are working while you're on this drug if you: Have kidney problems Have low blood pressure Take diuretics to treat high blood pressure Farxiga can cause serious cases of necrotizing fasciitis of the genital area (Fournier’s gangrene) in people with diabetes and low blood sugar when combined with insulin. SGLT2 Drugs for Heart Failure The big challenge now is for cardiologists to figure out when to prescribe this together with several other drugs in their appropriate doses for their patients with heart failure, Tang says. "This always happens with new drug indications, particularly when the drug cost can be relatively high.” A month's supply of Farxiga for diabetes costs $492. AstraZeneca expects the price to remain the same for heart failure treatment. The company says the amount people actually pay for the drug will depend on their health insurance coverage, their doctor, and any drug assistance programs or savings programs they use. A remaining question is whether other SGLT2 drugs will also help people who have heart failure. Studies are underway to figure that out. These trials may also shed some light on exactly how this group of drugs works to treat heart failure, and whether they're as effective in people with the other type of heart failure. https://www.webmd.com/heart-disease kalip
  10. I saw it on amazon and walmart but you should get at at health food stores
  11. Ingredients For the vegan mushroom stroganoff: 250 g pasta of choice , (vegan,) 1 cup vegetable broth 1 cup non-dairy milk, 1/4 cup all-purpose flour 1 teaspoon dried thyme leaves 1 teaspoon salt 1/4 teaspoon black pepper 2 tablespoons vegan butter 1 yellow onion, chopped 4 cloves garlic, minced 454 g brown or white button mushrooms sliced (about 6 cups sliced) For garnish: 1 handful fresh parsley, roughly chopped Parmegan (vegan parmesan) Instructions Bring a large pot of water to a boil and cook the pasta according to package directions. In a large measuring cup or medium bowl whisk together the vegetable broth, non-dairy milk, flour, dried thyme, salt, and pepper. Set aside. Melt the vegan butter in a large skillet or pot. When hot add the onion and garlic and sauté for about 5 minutes until the onion turns translucent and begins to brown. Reduce the heat to medium and add the mushrooms. Continue to cook for about 5 minutes until the mushrooms have softened and begin to release their juices. Pour in the broth and non-dairy milk mixture and continue to cook, stirring as needed for about 3 to 5 minutes until the sauce thickens. If the sauce gets too thick, add more vegetable broth or water if needed to thin. If the sauce is too thin, simply cook a little longer until you reach desired consistency. Add in the cooked pasta and toss well to combine. Divide among plates and serve, garnishing with parsley and Parmegan to taste. Nutrition Calories: 345 kcal | Carbohydrates: 60g | Protein: 13g | Fat: 6g | Saturated Fat: 1g | Sodium: 873mg | Potassium: 552mg | Fibre: 3g | Sugar: 5g | Vitamin A: 475IU | Vitamin C: 6.7mg | Calcium: 33mg | Iron: 2.2mg https://itdoesnttastelikechicken.com/recipes/ kalip
  12. CAN A PLANT-BASED DIET HELP WITH AUTOIMMUNE DISEASE A growing body of evidence indicates that a healthy plant-based diet can positively impact certain autoimmune diseases. In many cases, individuals report a significant reduction—or even total reversal—of autoimmune disease symptoms when they cut out meat and dairy. Autoimmune diseases are chronic illnesses that occur when healthy cells are destroyed by the body’s own immune system. There are more than eighty different types of autoimmune diseases, including type 1 diabetes, coeliac disease, lupus, psoriasis, and rheumatoid arthritis. Some studies, including YorkTest Laboratories 2017 survey, specifically highlighted animal products such as eggs and milk as aggravators of arthritis symptoms. According to a study published by the National Centre for Biotechnology Information, accumulating scientific evidence supports the health benefits of a vegetarian diet. “Both vegetarian and vegan diets typically emphasize vegetables, fruits, grains, legumes, and nuts,” says the study. “These results suggest that a vegan diet, with a high intake of fruits and vegetables and the elimination of animal products, could protect against the development of autoimmune conditions.” “In contrast, diets high in animal products and low in fibre might increase the risk of developing these autoimmune conditions,” adds the study. In general, red and processed meat consumption is increasingly linked to chronic health conditions such as cardiovascular disease, cancer, and diabetes. And consumers are turning to plant-based alternatives as a healthier option for themselves, the animals, and the planet. Plant-based whole foods are widely considered some of the most healthy. Rheumatoid Arthritis According to a review published in the scientific journal Frontiers in Nutrition, a plant-based diet can help alleviate the symptoms of rheumatoid arthritis. The review suggests that changes in diet “might play an important role in RA management and remission.” “Several studies have shown improvements in RA symptoms with diets excluding animal products,” said the review. “Studies have also shown that dietary fibre found in these plant-based foods can improve gut bacteria composition and increase bacterial diversity.” “Although some of the trigger foods in RA patients are individualized,” explains the review. “A vegan diet helps improve symptoms by eliminating many of these foods.” However, the study also notes that “further research is needed to test the effectiveness of plant-based diets.” Specifically how it impacts joint pain, inflammation, and the general quality of life in patients with RA. Review co-author Dr. Neal Barnard is also the founding president of the Physicians Committee for Responsible Medicine (PCRM), a plant-based advocacy group. And according to PCRM, a plant-based diet is effective in the reduction of arthritis symptoms in several ways. PCRM suggests that plant-based diets can help to reduce inflammation, swelling, and pain. It also suggests that they can help lower body mass index (BMI) and encourage healthy gut bacteria. Study co-author Hana Kahleova, MD, PhD, and the director of clinical research for PCRM, echoes this theory. “A plant-based diet comprised of fruits, vegetables, grains, and legumes may be tremendously helpful for those with rheumatoid arthritis,” says Kahleova. “This study offers hope that with a simple menu change, joint pain, swelling, and other painful symptoms may improve or even disappear.” Lupus Lupus is a long-term autoimmune disease with symptoms including inflammation and swelling. As with other autoimmune conditions, it occurs when the body’s immune system targets healthy tissue. It can also cause damage to a person’s joints, skin, and organs. As with rheumatoid arthritis, many of those with experience of lupus promote a plant-based diet to help alleviate symptoms. Brooke Goldner, MD, is the bestselling author of “Goodbye Autoimmune Disease and Goodbye Lupus” (2015). The now-celebrity doctor says that switching to a plant-based diet actually reversed her lupus diagnosis. She staunchly advocates for a plant-based diet and has inspired others to treat ongoing health conditions by changing their lifestyle. One person inspired by Goldner is the Australian writer and model Robyn Lawley, who recently announced that her lupus has improved to the point that she no longer needs medication. Lawley said that going vegan and following Goldner’s anti-inflammatory diet has helped her improve her condition. “I’m no doctor but this is what’s happening in my life,” explained Lawley on Instagram. “After being vegan for almost a year I’m just beyond happy that it’s helping me.” The Grammy-award winning singer Toni Braxton is also transitioning to a plant-based diet to help manage the symptoms of lupus. She recently told the healthy lifestyle magazine Prevention: “I’m becoming plant-based. I’m in the process. But I’m going fully plant-based. It’s making me feel better, so there is some truth to it.” Those diagnosed with lupus are generally encouraged to avoid foods that can cause inflammation. Some animal products—and dairy items, in particular—are linked to increased inflammation. Conversely, research suggests that plant-based alternatives can help to reduce inflammation and swelling. Multiple Sclerosis Multiple sclerosis (MS) is a lifelong condition, the symptoms of which can range from serious to mild. In the case of MS, the immune system destroys myelin—the fatty coating surrounding nerve fibres in the brain and spine. The primary symptom of MS is fatigue. But other potential symptoms include problems with vision and movement. Saray Stancic, MD, says that adopting a plant-based diet helped her treat her own MS. After nearly a decade of other treatment, Stancic was dependent on a walking stick and several medicines that caused difficult side effects. Adopting a plant-based diet had such a positive impact on her that she founded Stancic Health and Wellness in 2012, to help others treat chronic disease holistically. According to PCRM, there are several studies that indicate adopting a diet low in saturated fat may play a key role in MS management. Nutritionfacts.org, a plant-based advocacy group founded by Dr. Michael Greger M.D., echoes this belief. Roy Swank, an academic neurologist, first proposed a low-saturated-fat diet for the treatment of MS in 1949. According to Nutritionfacts, one study reported 95 percent of patients with early-stage MS experienced no progression in their disease 34 years after adopting a low saturated fat, meat, and dairy restricted diet. Some patients, who already displayed advanced symptoms, also experienced significant benefits from adopting the diet. The Irish author, public speaker, and vegan athlete Conor Devine is also vocal about the benefits of a plant-based diet for MS. In 2018, Devine spoke to LIVEKINDLY about how he overcame his MS symptoms and became a plant-powered athlete. “I have intermittent MS symptoms every day. But since I went plant-based my health has improved substantially,” he said. While others also support the benefits of a plant-based diet, Devine noted that his positive experience was personal to him. https://www.livekindly.co/ kalip
  13. Positivity Isn’t Naïve—It’s Exactly What the World Needs Today In times when people are struggling with their health, finances, loneliness, and tragic circumstances beyond their control, it might seem simplistic or patronising to encourage them to be more positive. Here’s why it’s crucial to stay positive right now. Have you heard of being a Pollyanna? It is a common way to label someone whose glass is always half full, despite any circumstance. The term came from a film about an irrepressibly optimistic young girl named Pollyanna. I’ve never seen the movie, but I do know that characters like Olaf from Frozen, Phoebe from Friends, or Kimmy Schmidt from The Unbreakable Kimmy Schmidt all share that Pollyanna brand of positivity. Like Pollyanna, people with high levels of optimism are often judged as immature or uneducated. But what if science supported that positivity is, in fact, the smarter way to be? Positivity 101 There is a strong body of evidence that the right kind of positive thinking can change the world. Let’s look at the biology, psychology, and wisdom in the practice of positivity. First, let’s consider what positivity is and is not. Positivity is about making an effort to experience and prolong positive emotions (the ones that feel good). It is also the ability to quickly recognise and move through negative emotions (the ones that feel yucky). Positivity is not pushing negative feelings away or pretending not to experience them. When you spend more time experiencing positive emotions rather than negative ones, you are said to be flourishing—or thriving. Biology—The Evolutionary Role of Negative Emotion Emotions and your ability to regulate them is what separates you and other humans from other species. All emotions serve a function. But until recently, researchers spent more time looking at the negative ones. The oldest part of your brain, sometimes called the reptilian brain, has the job of keeping you safe. This part of your biology is primitive and animalistic, and is responsible for the fight-or-flight-or-freeze response, which is designed to protect you from potential dangers. For example, we used to live in groups of around 150 people. This helped with our health and physical safety and the sharing of responsibilities. People would know everyone in their tribe. Our strong ability to pick out things that were different in our environment helped us to notice strangers who might carry disease or threaten our safety as well as other predators who posed a real threat. There was an important evolutionary reason for negative emotions. What has not been as clear to scientists is the role of positive emotions. Your emotional responses are like the gear system in a car. All gears are important, but the last thing you want is to get stuck in a gear that isn’t right for the type of driving you’re doing. When you get stuck in the emotional response loop, you might be making fear-based decisions about things that would not normally be scary. This is like being stuck in high gear while trying to get up a hill. Simply put, you need to be able to shift emotional gears when you need to. Psychology—What Are Positive Emotions? Emotions impact your relationships, how you make choices and decisions, what you pay attention to, and your personal identity. Positive emotions help with open-minded thinking; they build your resilience and have the power to undo the impact of negative emotions. Think of how strongly you can connect with someone over an inside joke, or how good you feel when you stand at the edge of a vast forest. Positive emotions have power. The following list contains many helpful positive emotions from Barbara Fredrickson, a leading expert from UNC-Chapel Hill and author of Positivity: Awe Gratitude Hope Inspiration Interest Joy Love Pride Serenity Examples of unhelpful emotions include the following: Anger Displeasure Fear Frustration Guilt Loneliness How Wisdom Practices Help While negative emotions demand attention, positive emotions tend to be more fleeting and subtle. Meditators often experience greater emotional atonement over time. This is good because, in order to keep your emotions working, you need to drive them as opposed to letting them take the wheel. Try these wisdom practices to help you balance out your emotions: Loving-Kindness Meditation It has been shown that loving-kindness meditations enhance interpersonal relationships and increase compassion. Even Charles Darwin, who is more commonly remembered as the mind behind the survival-of-the-fittest theory, believed that compassion was a key to flourishing communities according to his book Sociability. Meditation helps you to avoid rumination and to transform suffering. In meditation, the practice of present-moment awareness helps put space between emotion and reaction. Mindfulness Mindfulness is a path to become more aware of how you think. As you practice, you become more attuned to your emotional sensations and more equipped to catch yourself when your negative emotions take over. Gratitude Another wisdom practice for building positive emotions is gratitude. Giving thanks for the things and the people who surround you (or maybe who you can’t wait to see) is shown to lift moods and counteract depression and negativity. How to Become More Positive If you are considering that Pollyanna may have had it right and that positive emotions might help you to cope or to thrive during this current pandemic, here are a few ways to cultivate optimism. Create a positive bedtime ritual. Think of a time where you felt a very strong positive emotion and replay that story in your head as you fall asleep. Think of telling the story to someone who wasn’t there by recreating the physical environment, the physical sensations behind the emotion, and the emotional word you would use to describe the event. Look for opportunities to have positive experiences. Even though we cannot go outside, we can look through yearbooks, spend time looking at the stars, or have a call with an old friend. Schedule time in your day to create experiences designed to provoke positive emotions. Notice and name the negative. When a negative emotion happens, notice the sensation, name the emotion, then move through it as quickly as possible. Keep hope close at hand. Hope is the one emotion that can flip a negative to a positive almost instantly. In the words of The Stress-Proof Brain author and neuroplasticity expert, Melanie Greenburg, “Positive emotions and mental states may make people more resilient to stress, like sturdy tree branches that bend but don’t break when battered by a storm.” Optimism and positivity do help, and they have a contagious effect that can spread despite social distancing. https://chopra.com/articles kalip
  14. What Is the Best Self-Care during the COVID-19 Crisis? Self-care should be uppermost in our minds during the COVID-19 crisis, for several urgent reasons. Self-care returns a sense of control over your own life. It gives you an integrative approach to mind and body. It aligns you with the best knowledge currently available about who is more at risk for developing acute symptoms after being infected. Your immune status is complex, and in mainstream medicine, the chief determining factor is traditionally considered to be genetic. However, there are strong links to underlying low-level chronic inflammation connected to lifestyle that is found in most if not all common disorders including heart disease, type 2 diabetes, some cancers, and even obesity. COVID-19 has a mortality rate that increases with age and pre-existing conditions, as we all know by now. What is less publicised is that the hospitalisation rate, which is very high at over 15%, also affects younger age groups even though their death rates are lower. In those who are most susceptible, the virus creates an acute and severe form of inflammation referred to as a “cytokine storm”, which then leads to severe symptoms and respiratory damage, even death. Cytokines are the chief protein mediators of inflammation in the body. We can use this information about the dangers of having an elevated pre-existing state of low-level chronic inflammation to perhaps offset the possibility of the kind of acute inflammation that puts a person at risk for severe infection and hospitalisation. Let me emphasise the “perhaps” caution. The research on low-level chronic inflammation grows by the year and is very significant. You can go to many websites or our recent book “The Healing Self” to discover the kinds of foods and lifestyles that are either inflammatory or anti-inflammatory. But it is only in the field of integrative medicine that there is a cumulative understanding that anti-inflammation practices need to embrace mind and body. The general public, including mainstream doctors, are more often than not ill-informed of the research that connects meditation and yoga to the benefits of anti-stress, anti-inflammation, and anti-infection. Without a doubt, meditation and yoga have no side effects and are backed by decades of research over their benefits. To this has been added so-called “vagal breathing”, related to the ability of the vagus nerve to induce a relaxed state simply by doing regular deep breathing that equalises breathing in and breathing out (there are numerous websites providing instructions on this simple but effective technique, which is now a standard recommendation for countering stress). Biophysical anthropologist William C. Bushell points to the damage created by the body’s own immune defences. To quote a recent article at Medium.com by Maureen Seaberg, “Bushell says that inflammation is the primary way COVID-19 kills. ‘Spread of the virus through the body leads to widespread and intensive activation of the inflammatory defences throughout the body, though originally intended to combat the pathogen, but at this point instead resulting in widespread tissue damage, and fatally, to acute respiratory distress syndrome (ARDS), in which the lungs become flooded and respiratory failure ensues; the viral toxins themselves play a much lesser role in the tissue damage that ultimately can produce extreme critical disease states (pulmonary aspiration, septic shock), and potentially death.” The uncharted frontier is whether chronic inflammation, which is thought to be widely prevalent in our stress-filled, junk food eating, sleep-deprived society, increases the odds that the body might erupt into acute inflammation, e.g. a cytokine storm, when infected with viruses like COVID-19. One can think of this storm as a kind of over-reaction by our immune system. The big question is whether low-grade chronic inflammation may set the stage for this over-reaction and increase the odds of a “cytokine storm” in the presence of a nasty virus like COVID-19. Meanwhile, meditation, yoga, vagal breathing, and other relaxation techniques might provide an edge in the current crisis by reducing the chronic inflammatory state of the body. We’ve joined Bushell and other concerned figures, including Michelle Williams, S.D., Dean of Harvard’s School of Public Health, in advising that meditation and yoga (in addition to a healthy diet and ample sleep) be included in front-line efforts against COVID-19. The public is woefully uninformed about the mind-body benefits of these simple, effective measures, and every model of the pandemic foresees a huge overload on this country’s hospitals. The virus is more contagious and infectious than the flu, and it hospitalises and kills a much higher percentage of cases. The message about COVID-19 and inflammation is very important. It needs to be received by all of us for our common well-being at any time, but most urgently, now, we all must strive to reduce our base level of chronic inflammation. https://chopra.com/articles kalip
  15. Blood Clots Are Another Dangerous COVID-19 Mystery Hooman Poor, MD, was tired of watching his patients die, and it looked like another was slipping away. She was on a ventilator, but it wasn’t helping. Oxygen wasn’t getting to her organs. Acid was building in her blood. Her body was in shock. Her kidneys were failing, but he couldn’t put her on dialysis. She was too sick and probably wouldn’t survive it. Poor, a pulmonologist and critical care specialist at Mount Sinai Hospital in New York City, was about to call her family to deliver the crushing news. As he studied her lab results, he noticed that like many other COVID-19 patients, she had high levels of protein pieces called D-dimers, which are left over when the body breaks up blood clots. Her body was desperately trying, but failing, to clear blood clots, Poor believed. A Medical Mystery Around the world, doctors caring for COVID-19 patients have been trying to make sense of the same thing. When they draw blood from COVID patients, it clots in the tubes. When nurses insert catheters for kidney dialysis and IV lines to draw blood, the tubes quickly become clogged with clots. “Patients are making clots all over the place,” says Adam Cuker, MD, a hematologist and associate professor of medicine at the Hospital of the University of Pennsylvania. “That’s making management of these patients very challenging.” In addition to the well-known breathing problems, blood clots are a significant danger for COVID-19 patients. Clots are causing patients with COVID to have heart attacks and strokes; form strange rashes on their skin; and get red, swollen wounds that look like frostbite on their fingers and toes. On autopsy, the small vessels of the lungs and bowels, liver, and kidneys of COVID patients are choked with clots. With his patient desperately ill, Poor suspected he had nothing to lose, so he punted. “This is screaming blood clots. Why don’t we try tPA and see if it works?” he said. He gave his patient a powerful clot-busting drug that’s normally used to treat strokes. It’s risky. If used improperly, it can cause uncontrolled bleeding, which can be deadly. Within 30 minutes of getting the drug, his patient showed signs of improvement. Her carbon dioxide levels dropped, and other signs linked to shock seemed to improve. She lived for about another week, before ultimately dying. Poor has tried the drug on other COVID-19 patients, too. It hasn’t helped them survive, but he feels like it has shown him something about the disease. “It did bring to light the possibility that blood clots are playing a bigger role in this disorder than we previously appreciated,” he says. Lessons from the Dead Sharon Fox, MD, PhD, agrees with him. She’s a pathologist at Louisiana State University Health in New Orleans and has been doing autopsies on patients who have died with COVID-19. She’s finished 20 cases so far, and they share something in common: They are riddled with blood clots in the smallest vessels of the body. Lungs seem to be especially hard-hit. There, clots appear to have cut off blood flow to the small air sacs where blood cells would be exchanging oxygen and carbon dioxide. “There’s no ability for the blood to flow through and exchange oxygen like it should,” she says. Fox says the pattern of damage is striking. “I’ve never had a series of cases like this, where they all look the same, and all of the lungs have a similar pattern. There are types of vascular injury at autopsy that I haven’t seen before. I would say it’s new,” she says. Dangerous blood clotting is always a risk for critically ill patients. That’s especially true for those who are immobilized and on mechanical ventilators, as patients with COVID-19 often are. But a recent French study, which compared 150 patients with COVID-related respiratory failure who were treated in intensive care units to 145 patients who had respiratory failure, but were not infected with the new coronavirus, found significantly higher rates of blood clotting in the COVID patients. “We still need more controlled data, but based on clinical observations and the few studies that have been published, it looks like thrombosis [blood clotting] is more common in these patients,” Cuker says. Why blood congeals the way it does in some COVID patients is still an open question. One theory is that the body launches an immune attack called a cytokine storm to fight the virus that becomes self-directed, causing cells to kill themselves in an attempt to shut down the infection. Doctors believe that for some patients, the immune attack can end up doing the body more damage than the virus itself. This hyper-inflamed state is itself a well-known risk for blood clots. Cytokine storms can cause a condition called disseminated intravascular coagulation, or DIC, where patients both bleed uncontrollably and clot too much at the same time. Another possibility is that the virus may more directly cause the clotting. New Insights into ‘Why’ A research letter published this week in The Lancet reported evidence of viral bodies of the new coronavirus invading endothelial cells. The endothelium is the lining of our blood vessels. It directs important functions of the vascular system like clotting and swelling. Images captured with an electron microscope found traces of the coronavirus in endothelial cells in the heart, kidney, small bowel, and lung -- pretty much all over the body. Researchers collected the tissues during autopsies of three patients who died of COVID-19. Study co-author Mandeep Mehra, MD, medical director of the Brigham and Women’s Heart and Vascular Centre in Boston, says the findings suggest that the virus can directly infect the endothelium. He says that while COVID-19 can certainly cause breathing problems, he doesn’t think it’s just a lung disease. “This is actually a disease of the endothelium,” he says. Mehra says the infection starts in the lungs because breathing is the easiest way for the virus to enter the body. Once it infects the lung cells and begins to destroy them, it travels into the bloodstream. There, it infects endothelial cells, causing endotheliitis. Mehra thinks this endotheliitis comes from not only the direct infection of the blood vessel cells, but also from the haywire cytokine storm that the body launches to fight it off. “We’ve shown evidence of both.” He says this theory of infection explains some things that doctors have been trying to puzzle out. For example, certain conditions like high blood pressure, diabetes, and heart disease stress the endothelium. It’s no surprise, then, that people who have these conditions are also the ones who get the sickest when they catch COVID-19. It also helps to explain why patients have such low oxygen in their blood, but their lungs may not be as stiff as they typically are in patients who have respiratory distress with pneumonia. Mehra explains that one consequence of endotheliitis is that blood vessels can’t constrict the way they normally would. Typically, when a part of the lung becomes damaged, tiny blood vessels in that area close off so that blood will flow to a part of the lung that’s still working, where it can collect oxygen. This system protects the body from a sudden drop in oxygen, and it appears to break down in patients with severe COVID-19 infections. Mehra believes the infection of the endothelium is to blame. The bottom line, he says, is that clotting is a feature of the COVID-19 syndrome. When it becomes a big problem, the disease is advanced and very severe. For that reason, treating the resulting blood clots probably won’t work. He believes something worth trying might be to give patients drugs to support the endothelium, like ACE inhibitors and statins, along with anti-inflammatory drugs to tackle the cytokine storm, early in the course of the disease, but more research is needed to know for sure. If COVID-19 is really an endothelial infection, Mehra thinks that also helps to explain why ventilators aren’t helping more patients. A study of 5,700 patients hospitalized with COVID-19 in New York City found that while just 12% needed a ventilator to help them breathe, 88% of those patients died. “It’s not acting like influenza or other bacteria pneumonia where you get inflammation in the lungs and fluid buildup and very stiff lungs. That doesn’t seem to be the case, at least early on,” Poor says. “The virus is acting as if its primary target is the endothelium,” he says. That means the clots are just part of a much larger problem, one Poor says doctors don’t yet know how to solve. https://www.webmd.com/ kalip
  16. These articles may be relevant. https://www.sleepfoundation.org/press-release/national-sleep-foundation-recommends-new-sleep-times https://www.sciencedaily.com/releases/2008/07/080724123255.htm kalip
  17. COVID-19 May Mimic Heart Attack Signs in Some Eighteen patients with severe COVID-19 treated at a New York City hospital showed the classic signs of a heart attack on their electrocardiograms. But a closer look at each case revealed that more than half of these patients didn't have a blockage in a major artery, the typical trigger of a heart attack. Thirteen of the 18 patients died of cardiac causes while in the hospital, said a team from the New York University Grossman School of Medicine. These findings, published April 17 in the New England Journal of Medicine, suggest that there's something about the stress of severe COVID-19 that may be harming the heart in atypical ways, said one cardiologist who wasn't involved in the report. "As we continue to learn about the impact COVID-19 has to our heart, we have begun to identify unique and unusual effects," said Dr. Satjit Bhusri, a cardiologist at Lenox Hill Hospital, also in New York City. "Some patients present with abnormal EKGs, which can classically look as an acute heart attack, but without a true finding of a blocked artery." The real underlying cause for some cardiac deaths in these patients may be the stress, physical and otherwise, caused by COVID-19, Bhusri theorized. "This disparity is also seen in stress-induced heart disease, otherwise known as broken heart syndrome," he noted. The new report was led by Dr. Sripal Bangalore, a professor of medicine at NYU Langone Health. His team looked at the cases of 18 patients admitted with COVID-19 whose EKG readings indicated they had experienced a heart attack. The EKGs displayed a classic "elevated ST segment" reading that's indicative of heart attack, the doctors said. Ten patients displayed ST elevation at the time they were admitted to hospital, while eight others showed it during their hospital stay. However, based on coronary angiography scans and/or cath-lab investigations, 10 of the patients (56%) were found to have "non-coronary myocardial injury," Bangalore said. In layman's terms, that means a heart attack that was not caused by a blockage in an artery. So what might have caused the fatal or near-fatal heart injuries experienced by those 10 patients? It's not clear, but Bangalore's team said COVID-19 might injure the heart through a range of causes, including a rupturing of plaques in blood vessels, injury due to poor oxygenation, coronary spasm, tiny clots undetected by angiograms or more "direct" injury to vascular tissues. "This case series highlights the complexity in caring for patients with COVID-19 who have EKG changes suggestive of a heart attack," Bangalore noted. That's because using the standard heart treatment -- clot-busting drugs -- may sometimes be useless in these patients, as "half of them may not have major blockages," he explained. "Overall, the in-hospital death rate for these patients is very high and we urgently need studies to figure out how to best care for them," Bangalore said. Dr. Guy Mintz directs cardiovascular health at the Sandra Atlas Bass Heart Hospital in Manhasset, N.Y. Reading over the new report, he noted that the patients were "relatively young, with a median age of 63 years." The group had traditional heart risk factors: Two-thirds had high blood pressure, one-third had diabetes and 40% had high cholesterol. Mintz noted that similar findings were observed for Chinese COVID-19 patients who experienced heart crises during their care at hospitals in Wuhan, the original epicenter of the pandemic. Besides the formation of dangerous clots, severe COVID-19 "also stimulates the inflammatory system causing a cytokine storm -- an out-of-control inflammatory response to infection -- which can lead to more organ damage, whether it's the heart, lungs or kidneys," Mintz explained. https://consumer.healthday.com/ kalip
  18. How The COVID-19 Pandemic Will Change the Way We Live Large-scale social and behavioural changes — only some of which we can begin to predict — will follow the global catastrophe. The tsunami-like impact of a global pandemic has a way of drowning out foresight. Right now, it feels impossible to predict what the world will look like next week, let alone next year. Yet behavioural science and the broad sweep of history suggest that COVID-19 will transform our daily lives in the long run. The changes in progress — some predictable, others still hard to fathom — started brewing as soon as case counts began to escalate. An ongoing University of Southern California study published its first round of results in March, reporting that the coronavirus had already created significant shifts in people’s behaviour. Among the top findings: 85 percent of people reported washing their hands or using sanitizer more often than before, and 61 percent reported following social distancing guidelines. Twenty-two percent reported stockpiling essentials like food or water. Behavioural changes like these may be fear-driven to some extent, says Yale School of Medicine health psychologist Valeria Martinez-Kaigi, who is not affiliated with the study. Large numbers of respondents reported bracing for the possibility that they would lose their jobs or contract the virus. However, “fear-based behaviour modification is not proven to be sustainable,” Martinez-Kaigi says. After the initial coronavirus threat has passed, she predicts, new habits like hand washing, isolating and hoarding will scale back considerably. But that doesn’t mean they’ll disappear altogether. Survivors of collectively traumatic events tend to be especially vigilant in situations that burned them in the past. When people are forced to react to a deadly virus running rampant, they may be especially keen to keep future viruses at bay. Lessons from the Past The 1918 influenza pandemic, which killed about 675,000 people in the U.S., bent the arc of hygiene in lasting ways. In the following years, signs bearing the message “Spitting is Unlawful” sprouted up in places like train stations. Covering your cough grew to be considered common courtesy — before the pandemic, this etiquette was almost unheard of. In the wake of COVID-19, certain habits we’ve adapted will likely stick around as well, says Kate White, a behavioural scientist at the University of British Columbia. “Our vigilance around things like disinfecting surfaces — that’s probably going to continue,” she says. Our new ways of interacting with each other — “live long and prosper” salutes instead of handshakes, video chats instead of conference-room huddles — are also likely to stick to some degree. The old social norm is, “you meet someone, you automatically shake hands. Those social exchanges are going to evolve,” White says. “When you’re having meetings for work, people are going to start asking, ‘Do we have to meet in person?’ which is not something we would say before.” But what lingers most after a pandemic, or any large-scale catastrophe, is a pervasive sense that the world is fundamentally unpredictable — that life feels more fragile than it once did. The prospect theory of behaviour, popular among behavioural scientists and economists, states that we often give more weight to potential losses than gains in making a decision, and that we tend to overestimate the chances of an unlikely event like dying of a certain disease. Following a disaster, the tendency to believe that a threat is imminent may be magnified in some people. Those with more obsessive personalities, White says, might become hyper-avoidant in an attempt to construct a smaller, safer world for themselves. “Maybe they won’t go to certain social events. Maybe they won’t want to take the bus anymore.” Shifting Priorities A watershed event like this might also reorient broader attitudes about how the government should address public health issues. “If there’s anything that could underscore the interdependence, we all have, it’s this situation,” says Cornell University political scientist Suzanne Mettler. It’s possible, Mettler says, that our new embrace of this interdependence could drive us to demand a more supportive social safety net. If hundreds of thousands of Americans do die of COVID-19, and if people decide better health coverage could have prevented many of these deaths, a universal health care system might seem like an urgent necessity to more residents, she notes. Past pandemics have ushered in dramatic governmental shifts on this level. After British colonial authorities failed to protect India from the worst of the 1918 flu pandemic, the population’s fury helped fuel the Indian independence movement led by Mahatma Gandhi. It’s possible that fighting the coronavirus will inspire this kind of unity-seeking in the U.S. and elsewhere. “One would assume a pandemic would be something that could do that,” Mettler says. “This is, in many ways, like fighting a war. The enemy is the disease.” Mettler cautions that even in the wake of a pandemic, it will be difficult to dislodge the extreme polarization and mistrust that has divided Americans for years. White, however, sees the potential for some degree of post-pandemic solidarity, even if our philosophical differences persist. “People are appreciating that if we all act at the same time, we can see big changes,” she says. https://www.discovermagazine.com/health/ kalip
  19. Is 2 metres enough for social distancing? MIT researcher says droplets carrying coronavirus can travel up to 8 metres The novel coronavirus has prompted social distancing measures around the world. One researcher believes what's being done isn't enough Lydia Bourouiba, an associate professor at MIT, has researched the dynamics of exhalations (coughs and sneezes, for instance) for years at The Fluid Dynamics of Disease Transmission Laboratory and found exhalations cause gaseous clouds that can travel up to 27 feet (8.2 meters). Her research could have implications for the global COVID-19 pandemic, though measures called for by the Centres for Disease Control and Prevention and the World Health Organisation call for six and three feet (0.9 m and 1.8 m) of space, respectively. "There's an urgency in revising the guidelines currently being given by the WHO and the CDC on the needs for protective equipment, particularly for the frontline health care workers," Bourouiba told U.S. TODAY. Bourouiba's research calls for better measures to protect health care workers and, potentially, more distance from infected people who are coughing or sneezing. She said current guidelines are based on "large droplets" as the method of transmission for the virus and the idea that those large droplets can only go a certain distance. In a Journal of the American Medical Association article published last week, Bourouiba said peak exhalation speeds can reach 33 to 100 feet per second (36 km/h and 110 km/h) and "currently used surgical and N95 masks are not tested for these potential characteristics of respiratory emissions." The idea that droplets "hit a virtual wall and stop there and after that we are safe," is not based on evidence found in her research, Bourouiba said, and also not based on "evidence that we have about COVID transmission." Bourouiba argued that a "gaseous cloud" that can carry droplets of all sizes is emitted when a person coughs, sneezes or otherwise exhales. The cloud is only partially mitigated by sneezing or coughing into your elbow, she added. "In terms of the fluid regime—how the exhalations are emitted—the key point that we have shown is that there's a gaseous cloud that carries droplets of all sorts of sizes, not 'large' versus 'small' or 'droplets' versus 'aerosols,'" she said. Dr. Paul Pottinger, an infectious disease professor at the University of Washington School of Medicine, said questions remain about the distances at which the virus is effective. "For me, the question is not how far the germs can travel, but how far can they travel before they're no longer a threat. The smaller the germ particles, the lower the risk that they might infect somebody who would breathe them in or get them stuck in their nose or their mouth," Pottinger told U.S. TODAY. "The biggest threat—we think—with the coronavirus is actually the larger droplets. Droplets of saliva, snot, spit. Droplets that almost look like rain, if you will, when someone sneezes. Those droplets are large enough that gravity still acts on them. Usually, within about six feet of leaving somebody's body, those larger, more infectious droplets will drop to the ground. That's where the six-foot rule comes from." WHO referred to a recent scientific brief on the methods of transmission, which recommended "droplet and contact precautions for those people caring for COVID-19 patients." The CDC did not respond to an emailed request for comment. "WHO carefully monitors emerging evidence about this critical topic and will update this scientific brief as more information becomes available," WHO said in a statement. "WHO welcomes modelling studies, which are helpful for planning purposes. WHO teams work with several modelling groups to inform our work." If the coronavirus were effective at ranges of up to 27 feet (8.2 meters), as Bourouiba contends in her research, Pottinger said he believes more people would be sick. "It takes a certain number of viral particles, we call them 'virions,' or individual viruses, it takes a certain number of individual viruses to actually get a foothold inside the body and cause that infection to get going," he said. "Now, we don't know exactly what that number is, but it's probably more than a single virus. If you think about it, if this really travelled very efficiently by air, we wouldn't be having this conversation. Everybody would know it's true because everybody would be infected. If it was a 27-foot radius that was a high risk to somebody, this would be a totally different conversation. It's not." Bourouiba said she wants to see recommendations made based on current science not "policies based on supply, for example, because we don't have enough PPE (personal protective equipment)." It's well-known PPE is in short supply nationwide and health care workers have been desperately trying to find effective ways to deal with shortages. "Although there remains a lot of questions to be addressed about how much virus is at a given distance or not, we have no answer one way or another at this time," she said. "Therefore, the precautionary principle should drive the policies to state that we should have high-grade respirators used for health care workers." "Once that's decided, that's the thrust that's needed to now mobilise most effectively the kind of tremendous high production level that is possible to reach in a great country like the United States. This thrust is not happening." https://medicalxpress.com/ kalip
  20. Cholesterol: The Big Picture What Is Cholesterol? It's a waxy substance found in every cell of your body. You use it to make vitamin D and hormones like testosterone and oestrogen. It also helps digest food. Your liver makes cholesterol. Your diet also affects your cholesterol level. Cholesterol travels through your bloodstream in the form of tiny particles called lipoproteins. Low-density lipoprotein (LDL) brings cholesterol to cells throughout your body. It’s known as "bad" cholesterol because it can help plaque build-up in your arteries. This can make you more likely to have a stroke or heart attack. High-density lipoprotein (HDL), called "good" cholesterol, takes cholesterol back to the liver, where it's prepped to leave your body. Why Do Doctors Track It? If you have too much cholesterol in your bloodstream, it can stick to your artery walls, forming plaque. This is known as atherosclerosis, and it can partially or completely block blood flow, leading to problems such as angina or a mini-stroke. It can also make plaque more likely to rupture, leading to a clot that then blocks an artery, which can cause a heart attack and stroke. Atherosclerosis can take years to progress, and you may not have any symptoms until it's serious. That's why doctors recommend that once you reach the age of 20, you have your cholesterol levels tested at least every 4-6 years. You have high cholesterol if your LDL is 160 or above and your total cholesterol is 240 or above. (Total cholesterol is calculated using a formula: HDL levels + LDL levels + 20% of your triglyceride levels.) Having low HDL cholesterol -- less than 40 -- is also a risk factor. Keep in mind that your cholesterol levels are only part of the picture when it comes to heart health. Obesity, high blood pressure, diabetes, and smoking are all linked to artery disease, too. "Many different factors go into whether or not someone will develop heart disease, like genes, habits, and environment," says Karol Watson, MD, PhD, co-director of the UCLA Program in Preventive Cardiology, a professor of medicine/cardiology at the David Geffen School of Medicine at UCLA, and the director of the UCLA Barbra Streisand Women's Heart Health Program. You might hear about rare exceptions. Some people with high cholesterol and unhealthy lifestyles never have heart disease. But these cases don't mean you can dismiss your own high LDL levels, high blood pressure, smoking, or diabetes, says Jorge Plutzky, MD, director of preventive cardiology at Brigham and Women's Hospital/Harvard Medical School. Know that they can make you much more likely to have heart attacks and strokes. High cholesterol can run in families. And for most people, cholesterol levels rise steadily from age 20 to 65. But while you can't change your genes or your age, there are many things you can do to keep your cholesterol levels in check. For example, exercise regularly. (Being inactive can lower your HDL levels.) And if you're overweight, you're more likely to have higher LDL and lower HDL. Smoking also lowers your HDL levels and raises your LDL levels. Plus, "Our bodies use dietary saturated fat to make cholesterol," Watson says. LDL: Does Size Matter? In recent years, there's been some buzz about how the size of your LDL particles matters, with small particles being worse than larger ones. The theory is that smaller LDL particles "can get into the artery wall more easily," Plutzky says. "Then, once there, they're more prone to undergo changes that can cause heart attacks and strokes." Still, Plutzky notes that "for the average person, size of particles is not a definitive issue" and you probably don’t need to concern yourself with it. Watson agrees. "The smaller the particle, the worse it probably is, but even large, fluffy LDL is bad. All LDL is bad," she says. When Meds Might Help A healthy lifestyle is key. Some people also need medication. The most common cholesterol-lowering drugs are statins, and they can slash your risk of a heart attack or stroke. There are four groups of people who are candidates for statins: Those with LDL levels of 190 or higher Anyone who has had a cardiovascular event (like a heart attack or stroke) before Those with diabetes Anyone who, based on calculations, has a high risk of having a cardiovascular “event” (such as a heart attack) in the next 10 years. If you meet any of these criteria, it's not a good idea to delay treatment while you try making lifestyle changes first, Plutzky says. "It's not either-or. You can still make lifestyle changes," he says. But the statins will help bring down your numbers, "and extensive studies have shown them to be safe and well-tolerated." If you have low levels of HDL (“good” cholesterol), you should know that "none of the drugs that raise HDL have ever been shown to lower heart disease rates," Watson says. "So recommendations for raising HDL are to maintain a healthy weight, eat a Mediterranean-like diet, and exercise regularly." https://www.webmd.com/heart-disease kalip
  21. I'd prefer Coconut Oil, lots of ways on how it can be beneficial to one's health.
  22. This is a good read. Thank you Kalip.
  23. I am no expert on nutrition but this is what I read on nbcnews : Avocado Oil This recently popular oil has one of the highest smoke points, coming in around 271. °C This makes it ideal for an all-purpose oil or really high heat cooking. “Avocado oil is rich in monounsaturated fats, specifically oleic acid or omega-9, so it’s considered a heart-healthy oil with the potential to lower LDL (“bad”) cholesterol,” says Jackie Newgent, RDN, culinary nutritionist and author of “The All-Natural Diabetes Cookbook”. Newgent adds that you shouldn’t expect to get all of the same benefits of eating avocado by using just the oil. Yet, it is her oil of choice for making Freekeh “Fried Rice” and baked tortilla crisps. Coconut oil This controversial oil has been making waves in the nutrition scene due to its high saturated fat content. The American Heart Association recently advised against using coconut oil due its 82 percent saturated fat content. (In comparison, every other oil on this list only has about 10-20 percent saturated fat.) They cite a study that compared the effects of coconut oil, butter and safflower oil and found that butter and coconut oil raised LDL cholesterol, compared with safflower oil. Another study also found that coconut oil significantly increased LDL cholesterol compared with olive oil. With this in mind, if you do choose to cook with coconut oil, know that it has a smoke point of 175°C and a slight coconut flavour, making it the oil of choice for some to use in stir fries and pan frying. “If you're vegan, coconut oil provides some saturated fatty acids that you might not get from other foods, but I don’t recommend using coconut oil if you eat animal protein at all,”. https://www.nbcnews.com/
  24. Chocolate Loaf Cake Ingredients · 1/4 cup natural cocoa powder · 1 1/2 cups all-purpose flour · 1 1/4 cups granulated sugar* · 1 teaspoon baking soda · 1/2 teaspoon sea salt · 1/3 cup coconut oil, · 1/4 cup espresso, room temperature · 1 teaspoon vanilla extract · 1 teaspoon white vinegar · 1 cup water Directions 1. Preheat the oven to 170°C Line a 23 x 13 x 6cm loaf pan with parchment paper. 2. In a medium bowl, mix together the cocoa powder, flour, sugar, baking soda, and salt together. Set aside. 3. In a separate bowl, mix together the coconut oil, espresso, vanilla, vinegar, and water until well combined. 4. Stir in the wet ingredients into the dry ingredients and stir until just combined. 5. Pour the batter into the baking pan and bake for 50 - 55 minutes, or until a toothpick inserted in the middle comes out clean. https://cookienameddesire.com/recipes/ kalip
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